Horse

New Name for Immune-Mediated Myositis (IMM) Test

January 11, 2021
To more accurately reflect the genetic mechanism that underpins both immune-mediated myositis (IMM) and non-exertional rhabdomyolysis, the name of the VGL’s DNA test has been updated from Immune-mediated Myositis (IMM/MYH1) to myosin-heavy chain myopathy (MYHM).

VGL-led Team Identifies Genetic Variant Associated with Distichiasis in Friesians

December 04, 2020
An interdisciplinary team led by VGL Director Dr. Rebecca Bellone identified a genetic variant associated with distichiasis in Friesian horses. This is the first identification of a genetic variant linked to equine distichiasis, an ocular disease characterized by eyelashes that grow from abnormal positions along the edge of the eyelid and often lead to corneal irritation and damage.

Leopard Complex (LP) Linked to Increased Risk for Equine Recurrent Uveitis

July 16, 2020
Two recent studies by Veterinary Genetics Laboratory researchers and collaborators investigated equine recurrent uveitis (ERU) risk factors and genetic loci of interest in Appaloosas, including leopard complex (LP) and Appaloosa pattern-1 (PATN1), finding evidence that LP has an additive effect on ERU risk.

New Horse DNA Tests Launched

June 11, 2020
The Veterinary Genetics Laboratory has launched three new horse tests, one of which was researched and developed at the VGL.

Expanded Availability of Polysaccharide Storage Myopathy (PSSM1) Test

June 11, 2020
The Veterinary Genetics Laboratory is pleased to announce that it is now licensed to offer the Polysaccharide Storage Myopathy (PSSM1) test to the general public as both a standalone test and part of a genetic disease panel for Quarter Horses and related breeds.

VGL's Dr. Rebecca Bellone Featured in Equine Veterinary Journal Podcast

May 21, 2020
In early April, VGL Director Dr. Rebecca Bellone joined the Equine Veterinary Journal podcast "EVJ in Conversation" to discuss catastrophic breakdown in racehorses as well as her recent paper, which found no evidence of linkage between catastrophic breakdown and the mutation that causes Warmblood fragile foal syndrome type 1 (WFFS).