Half Arabian

Parentage/Genetic Marker Report

This DNA-based parentage test uses microsatellite marker analysis to compare the DNA profile of an offspring to the profiles of possible parents.

Horse Embryo Pre-Implantation Genetic Diagnosis

Pre-implantation genetic diagnosis (PGD) is a procedure used to screen embryos recovered after uterine flush to determine sex and genetic traits through DNA testing prior to implantation in the uterus.

Occipitoatlantoaxial Malformation (OAAM)

Occipitoatlantoaxial malformation (OAAM), an inherited developmental condition primarily found in Arabian horses, causes abnormal development of the vertebrae and results in compression of the upper cervical cord with subsequent neurological damage.

Severe Combined Immunodeficiency (SCID)

Severe combined immunodeficiency (SCID), an inherited condition primarily affecting Arabians, is characterized by an underdeveloped immune system that results in foals with elevated temperatures, respiratory stress, and diarrhea.

Lavender Foal Syndrome (LFS)

Lavender foal syndrome (LFS) is an inherited lethal coat color dilution found primarily in Arabian horses that is characterized by a dilute lavender, pale pink, or silver coat accompanied by severe neurological abnormalities.

Cerebellar Abiotrophy (CA)

Equine cerebellar abiotrophy (CA) is an inherited neurological condition found primarily in Arabian horses, and is characterized by neurological defects in foals including head tremors and ataxia.

Tobiano

Tobiano is a white spotting pattern characterized by white on the body that crosses the topline.

Red Factor

The extension gene, or red factor, determines whether a horse will have a chestnut base coat color or a black or bay base coat color.

Gray

The gray gene causes progressive depigmentation of the hair, often resulting in a color that is almost completely white by 6-8 years of age.

Dominant White Mutations – W5, W10, W20, and W22

Dominant white is a variable white spotting pattern caused by many different mutations in the KIT gene. The VGL tests for the four most common mutations known as W5, W10, W20, and W22. Homozygosity for W5, W10, or W22 is thought to be non-viable.