The Full Color/Pattern Panel combines both the coat color panel and the White Pattern Panel 2. This is the most comprehensive of the horse coat color/patterning panels offered by the VGL.
Panel can be purchased on MyVGL. See below for pricing and list of specific tests included in panel.
Note: Gray test included in this panel only detects presence or absence of gray. The panel report does not provide the number of copies (zygosity) of the gray gene. If zygosity information is desired, please order the individual gray test here.
Promotional pricing until December 31, 2021: $150 per animal
Dun is a coat color dilution characterized by lightening of the coat, with the head, lower legs, mane, and tail undiluted. Oftentimes, dun is also characterized by "primitive markings" such as a dark dorsal stripe, barring of the legs, shoulder stripes, and "cobwebbing" on the forehead.
The gray gene causes progressive depigmentation of the hair, often resulting in a color that is almost completely white by 6-8 years of age. The individual gray test will determine the number of copies of the gray allele (zygosity). The coat color panel tests detect presence or absence of the gray allele.
The silver dilution dilutes black/brown pigment to lighten the manes and tails of black and bay horses to a flaxen or silver gray. The silver dilution is also associated with an inherited ocular syndrome known as Multiple Congenital Ocular Anomalies (MCOA).
Splashed white is a variable white spotting pattern characterized by a broad blaze, extended white markings on the legs, variable white spotting on the belly, and often blue eyes. Six mutations in two different genes have been shown to cause the splashed white pattern.
Leopard complex or appaloosa spotting is a white pattern in horses characterized by a variable amounts of white in the coat with or without pigmented leopard spots. Horses homozygous for this variant have an ocular condition known as congenital stationary night blindness.
Dominant white is a variable white spotting pattern caused by many different mutations in the KIT gene. The VGL tests for the four most common mutations known as W5, W10, W20, and W22. Homozygosity for W5, W10, or W22 is thought to be non-viable.